Near the end of the article Dr. Stillwagon talks about the possible, and even more so likely, development of B-cell antibodies as a result of those spike proteins aimlessly floating around in your blood and/or whatever other parts of your body they might end up in... well regarding those B cells, I thought I'd add this here for anyone of you interested in the topic, I thought you'd marvel as well as I do at the complexity of just one small part of our immune system, only this production of different B cells, only this small part being so ingenious, and that is before we even consider the complexity of this system working in concert with all the varied T cell functions and a myriad of other functionality of our immune systems.
Now you can see why, when our bodies are presented with any kind of infection that they don't recognize, it takes our bodies days of these test mutations to find out how to start churning out antibodies that will be effective against the new infection.
And as you can see from the above video, if our immune system had long ago already created antibodies that would be effective against a current infection, our bodies' memory B cells will likely recognize the new infection due to its being similar enough to the previous infection (which is why many immunologists tried, while being canceled/censored, to tell us that people who had been previously infected with the first version of SARS some 20 years ago would already be equipped with memory B cells that would immediately recognize the new infection and as a result our body would be able to quickly produce effective antibodies against this new SARS and provide effective protection.
No, I'm not selling/promoting nor connected in any way with the author, I just bought this book a year or two ago and was spellbound, and still am.
I can't begin to describe to you to what stratospheric degree of appreciation for the wonder of your body you will find yourself climbing to after reading this book and better understanding the biology of your self.
Dr Kevin, can you take a look at his commentor? They are saying experts tried to say that peel who had pre existing cross reactivity to SARS would give protection.
However, on reality. Basic immunogky and 60 plus papers all make it clear that the IMPORTANT fact was that most of us had pre existing cross reactivity to COMMON COLD COROANVIRUSES.
OC43 and HKU1.
Isn't it weird that nobody knows this still? The research was all done. Repeatedly. But nobody learned it.
I'm now going to go through all comments, and I predict that even though you just explained how the killer T cells attack and destroy the organs and tissues transfected (the CLEAR AND PRESENT DANGER). I predict not one person will discuss this here.
My favourite part of COVID. People like you being told the mechanism mod harm in videos like Dr Kevin, and you idiots all complelty ignore it. Then run around pretending you know so much. And filling up comments spaces with everything EXCEPT the critical facts.
Pathetic.
Dr Kevin jist told you the MECHANSIM mfo harm. The most investigated topic in 5 years. And you ignored it. Just like all you CONSPIRACY idiots always do.
You've had 5 years to learn it, but you just keep ignoring.
Could the spikes that have been released and are just floating around, the bottom of the spike is “Sticky”, so would it be possible for some of them to stick onto the “castes”/clots that are being made by the endothelial damage?
Would you be able to talk to us about the clots?
Hirschmans analysis is making the rounds, it would be helpful if someone more knowledgeable, like you, to discuss them and maybe hirschmans analysis too?
I sometimes say the T cells go to the thymus to make their T cell army.
Cytotoxic CD8+ T Killer Cell Lymphocytes
The “T” stands for Thymus
Would you be able to talk about the CD4+ T helper cell that gets forgotten?
I agree with Will Thomson about Prof Arne BURKHARTs work.
The visual aid helped me quite a bit
You can see the “blue dots” “lymphocyte infiltration”
And the “brown ooze” Spike
He shows side by side pictures of what normal looks like vs DAMAGE.
I have listened to Will so much that I have the four common cold coronaviruses memorized
OC43
HKU1
NL63
229E
And he says
All the time
“Most of us had pre-existing cross reactivity to
Common cold corona viruses
OC43
HKU1”
Just like he has typed here
lol
Thank you again for talking about the cytotoxic t killer cell lymphocytes.
The spike proteins are glycoproteins and will tend to clump red blood cells together due to their stickiness, called hemagglutination. This can affect blood flow to certain areas of the body, resulting in ischemia, one of the first stages of tissue pathology. It can also cause transient ischemic attacks, brain fog, and sometimes strokes. As far as the rubbery clots are concerned, I think this is coming from misfolded proteins that arise from the frame shifting that occurs during the translation of the spike protein due to the pseudouridines that they put in the mRNA concoction. These misfolded proteins can be amyloid like and will not be broken down by enzymes, and therefore build up along vessel walls. Also, some people will get the IgG shift to type 4 after repeated booster shots, meaning the antibodies produced will not attach to the spike. This will allow the spike to attach to ACE2 receptors along vessel walls and the lining of the heart resulting in inflammation and myocarditis. I’ll be making a new video soon discussing T-cell cross reactivity and the different roles of the various types of T-cells.
Thank-you so very much for taking the time to reply.
“Foreign antigens are presented to T cells by antigen-presenting cells (APCs), like dendritic cells, macrophages, and B cells, which engulf the antigen, break it down into smaller peptides, and then display these peptides on their cell surface bound to major histocompatibility complex (MHC) molecules, allowing the T cell receptor (TCR) to recognize and bind to the antigen-MHC complex, triggering an immune response; essentially, T cells cannot directly recognize a free antigen, they need it to be presented in this complexed form on an APC. “
“MHC expression:
Both dendritic cells and macrophages express MHC class II molecules, which are necessary for presenting antigens to CD4+ helper T cells.”
Internet search:
“B cells can sometimes become "auto-activated" and produce antibodies that target the body's own proteins (self-antigens), leading to autoimmune diseases where the immune system attacks healthy tissues; this phenomenon is known as "autoimmunity.". “
The spikes get blown up along with the cell from the killer T cells…those spikes are just bits and pieces now along with the rest of the destroyed cell.
We also have the spikes that had gotten released from the cell
Free floating in the bloodstream.
T cells don’t recognize these free floating spikes but the B cells can
Internet search:
“Do B cells make autoantibodies?
Since autoantibodies are specific antibodies produced by B cells against self-antigens, these autoantibodies can bind to target antigens or target cells and organs, subsequently mediating cytotoxicity, inflammation and tissue damage.”
I was fishing when I said “sticky” because I was hoping you you talk about the transmembrane anchor and the S2 part of the spike.
Here is a 42 second video animation showing it.
See how it acts like a winch?
‘Model of Membrane Fusion by SARS-Cov-2 Spike Protein’
What the gpt says about the second mechansim .potential auto antibodies.
The relationship between mRNA vaccines and the production of autoantibodies by B cells has been the subject of several studies. Autoantibodies are antibodies that mistakenly target and react with an individual's own tissues or organs, potentially leading to autoimmune disorders.
A study published in Nature Communications found that mRNA-based COVID-19 vaccines do not promote the development of autoantibodies. The research indicated that vaccinated individuals did not exhibit new autoantibody responses post-vaccination, suggesting that mRNA vaccines do not trigger autoimmune reactions in healthy individuals.
Research from the Benaroya Research Institute reported that COVID-19 mRNA vaccination was not associated with the development of new autoantibody responses. This study supports the safety of mRNA vaccines concerning autoimmunity.
Studies Observing Autoantibody Production:
Conversely, a study in Vaccines noted the development of autoantibodies following the BNT162b2 mRNA COVID-19 vaccine. The researchers observed that a subset of vaccinated individuals developed autoantibodies, though the clinical significance of these findings requires further investigation.
Another study reported that mRNA-based anti-SARS-CoV-2 vaccines can induce the production of de novo antinuclear antibodies (ANA) in approximately 28.57% of subjects. The percentage of positivity appeared to correlate with the number of vaccine doses received.
Considerations for Individuals with Pre-existing Autoimmune Conditions:
For individuals with autoimmune diseases, such as systemic lupus erythematosus (SLE), studies have shown that mRNA vaccinations do not significantly increase disease activity. While some patients may experience mild to moderate flares post-vaccination, severe exacerbations are rare, and the benefits of vaccination outweigh the risks.
Conclusion:
The current body of literature presents mixed findings regarding the induction of autoantibodies by mRNA vaccines. While some studies report minimal to no autoantibody production post-vaccination, others have observed the development of autoantibodies in certain individuals. Importantly, the presence of autoantibodies does not necessarily equate to autoimmune disease, and further research is needed to understand the clinical implications fully. Overall, mRNA vaccines have been deemed safe and effective, with the benefits outweighing potential risks.
Thank you for sharing that. The amount and duration of exposure to foreign antigens or mRNA-encoded proteins does influence the risk of autoimmune reactions, and this has been researched and verified. We are at the tip of the iceberg on this as there is no way to know how many foreign antigens are produced nor how long they last. People are waking up all over the world. It's ultimately up to us as enlightened potential consumers to reject these dangerous products and encourage others to reject them as well.
Dr Kevin. Absolutely amazing work again , since you have now explained the killer T cells attack in clear and present danger. And now you said it again at the start of this video.
Now, I want you to always keenly observe your audience, and watch how ALL of them , do not aknowledge and demonstrate they understood what you said. I can. Cynthia Ross can.
100% of everyone else will ignore you, each and every time.
This has been going on the entire 5 years.
Excellent video, and I'm glad you've joined the few docotors warning and educating on the correct mechanism of harm.
You join, me, Dr bhakdi and burkhart, Dr John Campbell, Marc girardot and Bret weistien . We have all been repeating this. In all cases, everyone (99.99%) complelty ignore and never learn.
No scientific explanation hmfor why. It's just what's happening on our reality.
I'll add your video to my collection of doctors who can get it right.
Excellent work.
Having said that. Concerns about auto antibodies I strongly feel is a huge irrelevant distraction, from the clear and present danger of t cells destroying pekeks endothelium, brains hearts and organs. Which 99% of he planet still haven't learned yet.
Additionally. No docotrs ever explains what happens after antibodies stick.
So antibodies stick to stuff. So what? They don't do any damage. They just stick.
So then what happens? People are hugely ignorant of that.
My understanding is it triggers the Phagocyte activation pathway. The complement. System.
Yes, the first mechanism of harm is cytotoxic T-cell destruction of cells that are making foreign proteins coded by the injected mRNA. My point is, T-cells are not going to be able to stop all foreign proteins from being released into the bloodstream, and there are certainly downstream consequences, autoimmunity being one.
I know. I'm highly intelligent ex government epidemiologist and I've been focused on this topic for the entire 5 years. My mum had immature t cell issues. So I knew all about them.
So when they whole talent just "forgot" T cells and only talked antibodies, it be and clear something was very wrong.
This is the reality we are in. You are amazing and rare from explaining the first MECHANISM. T cells attack.
However, telling people. l is no use. 99.99% of your audience WILL NOT learn and repeat that. 5 other prominent doctors have tried for years before you, and they all failed too.
It is essential to focus on that main topic. While the auto antibody concept may be a side and additional potential issue. It is utterly irrelevant and distracting in terms of what everybody has been trying to comprehend for 5 years, and still can't fathom it.
Your audience will act like they comprehend. But you won't see them demonstrate they do. They won't use "killer T cells" or "cytotoxic T cells" or "lymphocytes" in their comments, ever. 1 in a 1000 can.
The first mechanism is proven in all the pathology, but doctors literally can't read and comprehend it. "Lymphocyte infiltration".
I don't see how the auto antibody will ever be adopted , proven or seen as a decisive issue. So I strongly advice not to focus on it. And would be great if you could do more clear videos discussing the first MECHANSIM, but add proof in your videos. Pathology biopsy history evidence that already exists. That might help a bit.
The fact that you can comprehend and talk t cells attack makes you 1 in a 1000 experts able to do that. Critically important! The next step is to observe and confirm that your audience can't learn and won't repeat. We need to expose and understand why they all can't learn it. Hypnosis or something weird?? Something metaphysical/supernatural is happening to us all.
Further to Dr. Stillwagon's response, there are many more hypothetical mechanisms of harm beyond autoimmunity and hijacked cell destruction. For example, (1) plasmid "contamination" related or reverse transcription related uptake of foreign genetic code in human cells, (2) spike protein interfering with DNA self-repair, (3) neurological damage related to neurons being hijacked (and self-attacked) and/or spike protein crossing the blood-brain barrier causing prion formation, (4) blockage of the vascular endothelium (high surface area target for cell hijack) and related micro-clotting. It seems that the more time passes, the more hypothetical mechanisms of harm are discovered. I propose there is nothing healthy in these shots, only damage. The only good shots are the placebos, which I also think are part of the attack.
Dr Kevin, please observe this commentor. As I said. They all avoid aknolwedging the very sp ific MECHANSIM for harm of auto immune ATTACK BY killer T lymphocytes. They generalise and say "autoimmunity" (not helpful).
This person has tried to muddy the water by calling this me hansi ma "hypothesis" when it is a demonstrated known fact.
Additionally, they say things like "other mechanisms of harm are the bad ualr blockages". They mens the mystery clots.
When the clots are not a mechanism of harm. They are the result of T cells endothelial attack and damage. Destroying endothelium, releases clotting factor, and the fibrin, cell debris, spike and immune cells all gunk up to make the clots. But hey all can't figure out what's triggering the clots, because they all won't learn and focus on "killer T cells attack" (Bhakdi) or "obliterative lymphocytic vasculitis"(Burkhart) . Or "carpet bombing of he endothelium" (GIRARDOT) .
They will all talk speculative avoidance and distracting nonsense until the cows come home. And never share and repeat "killer T cells attack and destroy cells". Ever.
Something's wrong with all their brains, I'm afraid.
I'm fine, and have been observing this lunacy in almost everyone for 5 years. Some of us are "normal".
They all avoid he topic, or try to be awkward and difficult. This way, the real fact , never becomes public knowledge.
That's why they are all stuck in a 4 year investigation going nowhere.
No, the proteins cannot be detected in a blood test, at least not yet... and whoever comes up with a reliable, inexpensive, quick way to do it will become a billionaire overnight. Right now the proteins are indirectly detected with antibody assays that are directed against the proteins. Researchers are working on ways to refine those antibody tests to determine if the proteins came from mRNA shots, and which ones specifically. We're not there yet.
The real issue is researchers need to be doing biopsies staining for the T cells attacking .
To prove the damage.
Like Dr burkhart did.
Lymphocyte infiltration
Burkhart did his work three years ago and blamed the T cells attack.
EVERYBDOY listened just invited that and went "spike protein damage!!!!".
They all complelty failed to learn that killer t cells attack.
Their brains can't do it.
And most docotrs can't either. They are in denial. They can't even find "lymphocyte infiltration" when it's in all the autopsies. Dr Kevin, if you give me you email, I can send you loads of patjolgy evidence.
Dr Kory at his clinic has a spike detect test done in AL that I think measures the spike... or is it something else? If I wasn't on the road I could check my notes from a podcast his partner did. Whatever it is, it wasn't cheap.
Near the end of the article Dr. Stillwagon talks about the possible, and even more so likely, development of B-cell antibodies as a result of those spike proteins aimlessly floating around in your blood and/or whatever other parts of your body they might end up in... well regarding those B cells, I thought I'd add this here for anyone of you interested in the topic, I thought you'd marvel as well as I do at the complexity of just one small part of our immune system, only this production of different B cells, only this small part being so ingenious, and that is before we even consider the complexity of this system working in concert with all the varied T cell functions and a myriad of other functionality of our immune systems.
Oh, that B cell function of developing antibodies: https://rumble.com/v2hblzg-dr.-chris-martenson-explains-the-beauty-of-our-immune-system.html
Now you can see why, when our bodies are presented with any kind of infection that they don't recognize, it takes our bodies days of these test mutations to find out how to start churning out antibodies that will be effective against the new infection.
And as you can see from the above video, if our immune system had long ago already created antibodies that would be effective against a current infection, our bodies' memory B cells will likely recognize the new infection due to its being similar enough to the previous infection (which is why many immunologists tried, while being canceled/censored, to tell us that people who had been previously infected with the first version of SARS some 20 years ago would already be equipped with memory B cells that would immediately recognize the new infection and as a result our body would be able to quickly produce effective antibodies against this new SARS and provide effective protection.
And anyone who finds these workings of the immune system interesting would surely find this book equally interesting: https://www.abebooks.com/servlet/SearchResults?cm_sp=SearchF-_-NullResults-_-Results&sts=t&tn=%22the%20song%20of%20the%20cell%22
No, I'm not selling/promoting nor connected in any way with the author, I just bought this book a year or two ago and was spellbound, and still am.
I can't begin to describe to you to what stratospheric degree of appreciation for the wonder of your body you will find yourself climbing to after reading this book and better understanding the biology of your self.
Dr Kevin, can you take a look at his commentor? They are saying experts tried to say that peel who had pre existing cross reactivity to SARS would give protection.
However, on reality. Basic immunogky and 60 plus papers all make it clear that the IMPORTANT fact was that most of us had pre existing cross reactivity to COMMON COLD COROANVIRUSES.
OC43 and HKU1.
Isn't it weird that nobody knows this still? The research was all done. Repeatedly. But nobody learned it.
I'm now going to go through all comments, and I predict that even though you just explained how the killer T cells attack and destroy the organs and tissues transfected (the CLEAR AND PRESENT DANGER). I predict not one person will discuss this here.
.
My favorite parts of Covid:
#16
That the vaccine injured are allergic to themselves.
.
My favourite part of COVID. People like you being told the mechanism mod harm in videos like Dr Kevin, and you idiots all complelty ignore it. Then run around pretending you know so much. And filling up comments spaces with everything EXCEPT the critical facts.
Pathetic.
Dr Kevin jist told you the MECHANSIM mfo harm. The most investigated topic in 5 years. And you ignored it. Just like all you CONSPIRACY idiots always do.
You've had 5 years to learn it, but you just keep ignoring.
Thank you for the clear explanation.
Hi Dr. Kevin Stillwagon,
Thank-you for the video.
Could the spikes that have been released and are just floating around, the bottom of the spike is “Sticky”, so would it be possible for some of them to stick onto the “castes”/clots that are being made by the endothelial damage?
Would you be able to talk to us about the clots?
Hirschmans analysis is making the rounds, it would be helpful if someone more knowledgeable, like you, to discuss them and maybe hirschmans analysis too?
I sometimes say the T cells go to the thymus to make their T cell army.
Cytotoxic CD8+ T Killer Cell Lymphocytes
The “T” stands for Thymus
Would you be able to talk about the CD4+ T helper cell that gets forgotten?
I agree with Will Thomson about Prof Arne BURKHARTs work.
The visual aid helped me quite a bit
You can see the “blue dots” “lymphocyte infiltration”
And the “brown ooze” Spike
He shows side by side pictures of what normal looks like vs DAMAGE.
I have listened to Will so much that I have the four common cold coronaviruses memorized
OC43
HKU1
NL63
229E
And he says
All the time
“Most of us had pre-existing cross reactivity to
Common cold corona viruses
OC43
HKU1”
Just like he has typed here
lol
Thank you again for talking about the cytotoxic t killer cell lymphocytes.
The spike proteins are glycoproteins and will tend to clump red blood cells together due to their stickiness, called hemagglutination. This can affect blood flow to certain areas of the body, resulting in ischemia, one of the first stages of tissue pathology. It can also cause transient ischemic attacks, brain fog, and sometimes strokes. As far as the rubbery clots are concerned, I think this is coming from misfolded proteins that arise from the frame shifting that occurs during the translation of the spike protein due to the pseudouridines that they put in the mRNA concoction. These misfolded proteins can be amyloid like and will not be broken down by enzymes, and therefore build up along vessel walls. Also, some people will get the IgG shift to type 4 after repeated booster shots, meaning the antibodies produced will not attach to the spike. This will allow the spike to attach to ACE2 receptors along vessel walls and the lining of the heart resulting in inflammation and myocarditis. I’ll be making a new video soon discussing T-cell cross reactivity and the different roles of the various types of T-cells.
Hi Dr. Kevin Stillwagon
Thank-you so very much for taking the time to reply.
“Foreign antigens are presented to T cells by antigen-presenting cells (APCs), like dendritic cells, macrophages, and B cells, which engulf the antigen, break it down into smaller peptides, and then display these peptides on their cell surface bound to major histocompatibility complex (MHC) molecules, allowing the T cell receptor (TCR) to recognize and bind to the antigen-MHC complex, triggering an immune response; essentially, T cells cannot directly recognize a free antigen, they need it to be presented in this complexed form on an APC. “
“MHC expression:
Both dendritic cells and macrophages express MHC class II molecules, which are necessary for presenting antigens to CD4+ helper T cells.”
Internet search:
“B cells can sometimes become "auto-activated" and produce antibodies that target the body's own proteins (self-antigens), leading to autoimmune diseases where the immune system attacks healthy tissues; this phenomenon is known as "autoimmunity.". “
The spikes get blown up along with the cell from the killer T cells…those spikes are just bits and pieces now along with the rest of the destroyed cell.
We also have the spikes that had gotten released from the cell
Free floating in the bloodstream.
T cells don’t recognize these free floating spikes but the B cells can
Internet search:
“Do B cells make autoantibodies?
Since autoantibodies are specific antibodies produced by B cells against self-antigens, these autoantibodies can bind to target antigens or target cells and organs, subsequently mediating cytotoxicity, inflammation and tissue damage.”
I was fishing when I said “sticky” because I was hoping you you talk about the transmembrane anchor and the S2 part of the spike.
Here is a 42 second video animation showing it.
See how it acts like a winch?
‘Model of Membrane Fusion by SARS-Cov-2 Spike Protein’
https://youtu.be/e2Qi-hAXdJo?si=YycRJR5Pk5oDCHJ7
Thank you so very much
My most humble apologies for typing about clots, sorry about that, thank you for kindly answering.
What the gpt says about the second mechansim .potential auto antibodies.
The relationship between mRNA vaccines and the production of autoantibodies by B cells has been the subject of several studies. Autoantibodies are antibodies that mistakenly target and react with an individual's own tissues or organs, potentially leading to autoimmune disorders.
Studies Indicating Minimal Autoantibody Production:
A study published in Nature Communications found that mRNA-based COVID-19 vaccines do not promote the development of autoantibodies. The research indicated that vaccinated individuals did not exhibit new autoantibody responses post-vaccination, suggesting that mRNA vaccines do not trigger autoimmune reactions in healthy individuals.
Research from the Benaroya Research Institute reported that COVID-19 mRNA vaccination was not associated with the development of new autoantibody responses. This study supports the safety of mRNA vaccines concerning autoimmunity.
Studies Observing Autoantibody Production:
Conversely, a study in Vaccines noted the development of autoantibodies following the BNT162b2 mRNA COVID-19 vaccine. The researchers observed that a subset of vaccinated individuals developed autoantibodies, though the clinical significance of these findings requires further investigation.
Another study reported that mRNA-based anti-SARS-CoV-2 vaccines can induce the production of de novo antinuclear antibodies (ANA) in approximately 28.57% of subjects. The percentage of positivity appeared to correlate with the number of vaccine doses received.
Considerations for Individuals with Pre-existing Autoimmune Conditions:
For individuals with autoimmune diseases, such as systemic lupus erythematosus (SLE), studies have shown that mRNA vaccinations do not significantly increase disease activity. While some patients may experience mild to moderate flares post-vaccination, severe exacerbations are rare, and the benefits of vaccination outweigh the risks.
Conclusion:
The current body of literature presents mixed findings regarding the induction of autoantibodies by mRNA vaccines. While some studies report minimal to no autoantibody production post-vaccination, others have observed the development of autoantibodies in certain individuals. Importantly, the presence of autoantibodies does not necessarily equate to autoimmune disease, and further research is needed to understand the clinical implications fully. Overall, mRNA vaccines have been deemed safe and effective, with the benefits outweighing potential risks.
Dr. Stillwagon you have to listen to when the swedish minister of social affairs makes a fool of himself when he calls autoimmunity from the covid modRNA injections a conspiracy theory. The parliamentary debate is with english subtitlles in this link. https://patientmaktpatientcv.substack.com/p/sweden-parliamentary-debate-dna-contamination?utm_source=publication-search
Thank you for sharing that. The amount and duration of exposure to foreign antigens or mRNA-encoded proteins does influence the risk of autoimmune reactions, and this has been researched and verified. We are at the tip of the iceberg on this as there is no way to know how many foreign antigens are produced nor how long they last. People are waking up all over the world. It's ultimately up to us as enlightened potential consumers to reject these dangerous products and encourage others to reject them as well.
Dr Kevin. Absolutely amazing work again , since you have now explained the killer T cells attack in clear and present danger. And now you said it again at the start of this video.
Now, I want you to always keenly observe your audience, and watch how ALL of them , do not aknowledge and demonstrate they understood what you said. I can. Cynthia Ross can.
100% of everyone else will ignore you, each and every time.
This has been going on the entire 5 years.
Excellent video, and I'm glad you've joined the few docotors warning and educating on the correct mechanism of harm.
You join, me, Dr bhakdi and burkhart, Dr John Campbell, Marc girardot and Bret weistien . We have all been repeating this. In all cases, everyone (99.99%) complelty ignore and never learn.
No scientific explanation hmfor why. It's just what's happening on our reality.
I'll add your video to my collection of doctors who can get it right.
Excellent work.
Having said that. Concerns about auto antibodies I strongly feel is a huge irrelevant distraction, from the clear and present danger of t cells destroying pekeks endothelium, brains hearts and organs. Which 99% of he planet still haven't learned yet.
Additionally. No docotrs ever explains what happens after antibodies stick.
So antibodies stick to stuff. So what? They don't do any damage. They just stick.
So then what happens? People are hugely ignorant of that.
My understanding is it triggers the Phagocyte activation pathway. The complement. System.
Both mechanisms described would be highly dangerous.
Please demonstrated you understood the first MECHANSIM.
Thanks.
The entire planet is still trying to learn what the MECHANSIM of harm is.
You were just told it.
What is it?
Can you repeat it, using correct terminology?
Yes, the first mechanism of harm is cytotoxic T-cell destruction of cells that are making foreign proteins coded by the injected mRNA. My point is, T-cells are not going to be able to stop all foreign proteins from being released into the bloodstream, and there are certainly downstream consequences, autoimmunity being one.
I know. I'm highly intelligent ex government epidemiologist and I've been focused on this topic for the entire 5 years. My mum had immature t cell issues. So I knew all about them.
So when they whole talent just "forgot" T cells and only talked antibodies, it be and clear something was very wrong.
This is the reality we are in. You are amazing and rare from explaining the first MECHANISM. T cells attack.
However, telling people. l is no use. 99.99% of your audience WILL NOT learn and repeat that. 5 other prominent doctors have tried for years before you, and they all failed too.
It is essential to focus on that main topic. While the auto antibody concept may be a side and additional potential issue. It is utterly irrelevant and distracting in terms of what everybody has been trying to comprehend for 5 years, and still can't fathom it.
Your audience will act like they comprehend. But you won't see them demonstrate they do. They won't use "killer T cells" or "cytotoxic T cells" or "lymphocytes" in their comments, ever. 1 in a 1000 can.
The first mechanism is proven in all the pathology, but doctors literally can't read and comprehend it. "Lymphocyte infiltration".
I don't see how the auto antibody will ever be adopted , proven or seen as a decisive issue. So I strongly advice not to focus on it. And would be great if you could do more clear videos discussing the first MECHANSIM, but add proof in your videos. Pathology biopsy history evidence that already exists. That might help a bit.
The fact that you can comprehend and talk t cells attack makes you 1 in a 1000 experts able to do that. Critically important! The next step is to observe and confirm that your audience can't learn and won't repeat. We need to expose and understand why they all can't learn it. Hypnosis or something weird?? Something metaphysical/supernatural is happening to us all.
"Concept specific dementia"(?)
Further to Dr. Stillwagon's response, there are many more hypothetical mechanisms of harm beyond autoimmunity and hijacked cell destruction. For example, (1) plasmid "contamination" related or reverse transcription related uptake of foreign genetic code in human cells, (2) spike protein interfering with DNA self-repair, (3) neurological damage related to neurons being hijacked (and self-attacked) and/or spike protein crossing the blood-brain barrier causing prion formation, (4) blockage of the vascular endothelium (high surface area target for cell hijack) and related micro-clotting. It seems that the more time passes, the more hypothetical mechanisms of harm are discovered. I propose there is nothing healthy in these shots, only damage. The only good shots are the placebos, which I also think are part of the attack.
Dr Kevin, please observe this commentor. As I said. They all avoid aknolwedging the very sp ific MECHANSIM for harm of auto immune ATTACK BY killer T lymphocytes. They generalise and say "autoimmunity" (not helpful).
This person has tried to muddy the water by calling this me hansi ma "hypothesis" when it is a demonstrated known fact.
Additionally, they say things like "other mechanisms of harm are the bad ualr blockages". They mens the mystery clots.
When the clots are not a mechanism of harm. They are the result of T cells endothelial attack and damage. Destroying endothelium, releases clotting factor, and the fibrin, cell debris, spike and immune cells all gunk up to make the clots. But hey all can't figure out what's triggering the clots, because they all won't learn and focus on "killer T cells attack" (Bhakdi) or "obliterative lymphocytic vasculitis"(Burkhart) . Or "carpet bombing of he endothelium" (GIRARDOT) .
They will all talk speculative avoidance and distracting nonsense until the cows come home. And never share and repeat "killer T cells attack and destroy cells". Ever.
Something's wrong with all their brains, I'm afraid.
I'm fine, and have been observing this lunacy in almost everyone for 5 years. Some of us are "normal".
They all avoid he topic, or try to be awkward and difficult. This way, the real fact , never becomes public knowledge.
That's why they are all stuck in a 4 year investigation going nowhere.
Aspirin therapy? Time restricted eating? Curcumin? Can these proteins be seen on a blood test?
No, the proteins cannot be detected in a blood test, at least not yet... and whoever comes up with a reliable, inexpensive, quick way to do it will become a billionaire overnight. Right now the proteins are indirectly detected with antibody assays that are directed against the proteins. Researchers are working on ways to refine those antibody tests to determine if the proteins came from mRNA shots, and which ones specifically. We're not there yet.
The real issue is researchers need to be doing biopsies staining for the T cells attacking .
To prove the damage.
Like Dr burkhart did.
Lymphocyte infiltration
Burkhart did his work three years ago and blamed the T cells attack.
EVERYBDOY listened just invited that and went "spike protein damage!!!!".
They all complelty failed to learn that killer t cells attack.
Their brains can't do it.
And most docotrs can't either. They are in denial. They can't even find "lymphocyte infiltration" when it's in all the autopsies. Dr Kevin, if you give me you email, I can send you loads of patjolgy evidence.
Dr Kory at his clinic has a spike detect test done in AL that I think measures the spike... or is it something else? If I wasn't on the road I could check my notes from a podcast his partner did. Whatever it is, it wasn't cheap.